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View ResearchExplore the identification of potential inflammation markers for the outgrowth of cow's milk allergy (CMA). This study utilizes the Olink® Target 96 Inflammation panel to analyze saliva samples from infants with CMA, revealing significant differences in immune markers between those who outgrew their allergy and those with persistent CMA. The findings suggest TNF signaling and cytokine-cytokine receptor interaction pathways play crucial roles in allergy resolution.
Potential Inflammation Markers for CMA Outgrowth
Identifying key inflammation markers associated with cow's milk allergy outgrowth in children.
Saliva Proteins as Indicators of CMA Resolution
Analyzing saliva proteins to find immunological markers for resolving cow's milk allergy in infants.
Role of IL-15RA and CASP-8 in Persistent CMA
Increased IL-15RA and CASP-8 levels linked to persistent cow's milk allergy in infants.
KEGG Pathway Analysis in CMA Study
Using KEGG pathway analysis to identify important signaling pathways in CMA resolution and persistence.
Effects of Specialized Infant Formulas on CMA
Investigating the impact of amino acid-based and synbiotic-supplemented formulas on cow's milk allergy resolution.
TNF and TLR Signaling in CMA Resolution
TNF and Toll-like receptor signaling pathways play crucial roles in the resolution of cow's milk allergy.
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